Neutrophils, once considered simple foot soldiers of innate immunity, are now recognized as complex players in cancer. This review highlights how these cells, which typically defend the body from infection, can paradoxically support tumor progression through promoting angiogenesis, immune suppression, and metastasis. Yet, they also possess tumor-fighting capabilities under certain conditions. The dual nature of tumor-associated neutrophils (TANs)—either aiding or attacking cancer—depends on
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